Valproate Therapy Depresses Free Radical Scavenging Enzyme Activity: A Mechanism for Induction of Acute Pancreatitis or Hepatotoxicity

C.E. Pippenger; Frederick Van Lente; A. David Rothner; Xianzhong Meng

Departments

Valproate Therapy Depresses Free Radical Scavenging Enzyme Activity: A Mechanism for Induction of Acute Pancreatitis or Hepatotoxicity

C.E. Pippenger, Frederick Van Lente, A. David Rothner and Xianzhong Meng

Cleveland Clinic Journal of Medicine February 1989, 56 (6 suppl part 2) S-287-S-288;

C.E. Pippenger

Find this author on Google Scholar
Find this author on PubMed
Search for this author on this site

Frederick Van Lente

Find this author on Google Scholar
Find this author on PubMed
Search for this author on this site

A. David Rothner

The Cleveland Clinic Foundation, Cleveland, OH

Find this author on Google Scholar
Find this author on PubMed
Search for this author on this site

Xianzhong Meng

Institute of Keshan Disease, Harbrin Medical University, People’s Republic of China

Find this author on Google Scholar
Find this author on PubMed
Search for this author on this site

Article
Info & Metrics
PDF

This is a PDF-only article. The first page of the PDF of this article appears above.

PreviousNext

In this issue

Cleveland Clinic Journal of Medicine

Vol. 56, Issue 6 suppl part 2

1 Feb 1989

Table of Contents
Table of Contents (PDF)
Index by author

Download PDF

Article Alerts

User Name *

Password *

Email *

Email Article

Thank you for your interest in spreading the word on Cleveland Clinic Journal of Medicine.

NOTE: We only request your email address so that the person you are recommending the page to knows that you wanted them to see it, and that it is not junk mail. We do not capture any email address.

Your Email *

Your Name *

Send To *

Enter multiple addresses on separate lines or separate them with commas.

You are going to email the following Valproate Therapy Depresses Free Radical Scavenging Enzyme Activity: A Mechanism for Induction of Acute Pancreatitis or Hepatotoxicity

Message Subject (Your Name) has sent you a message from Cleveland Clinic Journal of Medicine

Message Body (Your Name) thought you would like to see the Cleveland Clinic Journal of Medicine web site.

Your Personal Message

CAPTCHA

This question is for testing whether or not you are a human visitor and to prevent automated spam submissions.

Citation Tools

Valproate Therapy Depresses Free Radical Scavenging Enzyme Activity: A Mechanism for Induction of Acute Pancreatitis or Hepatotoxicity

C.E. Pippenger, Frederick Van Lente, A. David Rothner, Xianzhong Meng

Cleveland Clinic Journal of Medicine Feb 1989, 56 (6 suppl part 2) S-287-S-288;

Citation Manager Formats

BibTeX
Bookends
EasyBib
EndNote (tagged)
EndNote 8 (xml)
Medlars
Mendeley
Papers
RefWorks Tagged
Ref Manager
RIS
Zotero

Valproate Therapy Depresses Free Radical Scavenging Enzyme Activity: A Mechanism for Induction of Acute Pancreatitis or Hepatotoxicity

C.E. Pippenger, Frederick Van Lente, A. David Rothner, Xianzhong Meng

Cleveland Clinic Journal of Medicine Feb 1989, 56 (6 suppl part 2) S-287-S-288;

Share This Article:

Tweet Widget
Facebook Like
Linkedin Share Button

Jump to section

Article
Info & Metrics
PDF

No related articles found.

Google Scholar

Cited By...

No citing articles found.

Google Scholar

Departments

When should we consider SGLT-2 inhibitors in patients with acute decompensated heart failure?
Our Peer-Reviewers for 2023
Our Peer-Reviewers For 2022

Show more Departments

Abstracts

Temporary Interruption of Benzodiazepines for intractable Childhood Epilepsy
ACTH Therapy in Infantile Spasms: Effects on Cerebrospinal Amino Acids, HVA, 5-Hydroxyin Dole Acetic Acid, and Histamine
Disseminated Cytomegalovirus in a Patient with Infantile Spasms Treated with ACTH

Show more Abstracts

Main menu

User menu

Search

Valproate Therapy Depresses Free Radical Scavenging Enzyme Activity: A Mechanism for Induction of Acute Pancreatitis or Hepatotoxicity

In this issue

Citation Manager Formats

Related Articles

Cited By...

More in this TOC Section

Departments

Abstracts

Similar Articles

Navigate

Authors & Reviewers