User profiles for Maria A. Nagel

Maria Nagel

Associate Professor of Neurology and Ophthalmology, University of Colorado School of …
Verified email at ucdenver.edu
Cited by 7115
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Varicella zoster virus vasculopathies: diverse clinical manifestations, laboratory features, pathogenesis, and treatment

…, RJ Cohrs, R Mahalingam, MA Nagel - The Lancet …, 2009 - thelancet.com
Vasculopathies caused by varicella zoster virus (VZV) are indicative of a productive virus
infection in cerebral arteries after either reactivation of VZV (shingles) or primary infection (…
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Varicella zoster virus infection: clinical features, molecular pathogenesis of disease, and latency

…, DH Gilden, RJ Cohrs, R Mahalingam, MA Nagel - Neurologic clinics, 2008 - Elsevier
Varicella zoster virus (VZV) is an exclusively human neurotropic alphaherpesvirus. Primary
infection causes varicella (chickenpox), after which virus becomes latent in cranial nerve …
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Adventitial fibroblasts induce a distinct proinflammatory/profibrotic macrophage phenotype in pulmonary hypertension

…, M Li, SS Pullamsetti, R Savai, MA Nagel… - The Journal of …, 2014 - journals.aai.org
KCEK and KRS conceived the study, delineated the hypotheses, designed the study, analyzed
and interpreted data, and prepared the manuscript; SRR, HKE, ALA, MGF, ML, SSP, SCP…
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Neurological disease produced by varicella zoster virus reactivation without rash

D Gilden, RJ Cohrs, R Mahalingam, MA Nagel - Varicella-zoster virus, 2010 - Springer
Reactivation of varicella zoster virus (VZV) from latently infected human ganglia usually
produces herpes zoster (shingles), characterized by dermatomal distribution pain and rash. …
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Analysis of human alphaherpesvirus microRNA expression in latently infected human trigeminal ganglia

JL Umbach, MA Nagel, RJ Cohrs, DH Gilden… - Journal of …, 2009 - Am Soc Microbiol
ABSTRACT Analysis of cells infected by a wide range of herpesviruses has identified
numerous virally encoded microRNAs (miRNAs), and several reports suggest that these viral …
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Prevalence and distribution of VZV in temporal arteries of patients with giant cell arteritis

…, D Chatelain, K Keyvani, W Brueck, MA Nagel - Neurology, 2015 - AAN Enterprises
Objective: Varicella-zoster virus (VZV) infection may trigger the inflammatory cascade that
characterizes giant cell arteritis (GCA). Methods: Formalin-fixed, paraffin-embedded GCA-…
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Neurological complications of varicella zoster virus reactivation

MA Nagel, D Gilden - Current opinion in neurology, 2014 - journals.lww.com
… Salazar R, Russman AN, Nagel MA, et al. Varicella zoster virus ischemic optic
neuropathy and subclinical temporal artery involvement. Arch Neurol 2011; 68:517–520 …
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Update on varicella zoster virus vasculopathy

MA Nagel, D Gilden - Current infectious disease reports, 2014 - Springer
Primary infection of humans with varicella zoster virus (VZV) causes varicella (chickenpox),
after which the virus becomes latent in cranial nerve ganglia, dorsal root ganglia and …
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Analysis of varicella-zoster virus in temporal arteries biopsy positive and negative for giant cell arteritis

MA Nagel, T White, N Khmeleva, A Rempel… - JAMA …, 2015 - jamanetwork.com
Importance Giant cell arteritis (GCA) is the most common systemic vasculitis in elderly
individuals. Diagnosis is confirmed by temporal artery (TA) biopsy, although biopsy results are …
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[PDF] psu.edu

Complications of varicella zoster virus reactivation

MA Nagel, D Gilden - Current treatment options in neurology, 2013 - Springer
Opinion statement Varicella zoster virus (VZV) is an exclusively human neurotropic
alphaherpesvirus. Primary infection causes varicella (chickenpox), after which virus becomes latent …
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