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II. Articles and Features of General Interest

Severe congenital tricuspid regurgitation in the newborn

A rare form with myxomatous valvular incomplete development

Douglas S. Moodie, M.D., Richard Sterba, M.D. and Norman B. Ratliff, M.D.
Cleveland Clinic Journal of Medicine September 1985, 52 (3) 403-408;
Douglas S. Moodie
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Richard Sterba
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Norman B. Ratliff
Departments of Cardiology and Pathology, The Cleveland Clinic Foundation
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ABSTRACT

A case of congenital tricuspid insufficiency in the newborn secondary to myxomatous degeneration of the tricuspid valve is described. There were myxomatous changes of the pulmonary and aortic valves. The mitral valve was normal. Histologic study showed an increased amount of glycosaminoglycans and a decreased amount of mature collagen of the tricuspid, aortic, and pulmonary valves. The etiology of congenital tricuspid insufficiency of the newborn can be expanded to include failure of the tricuspid valve leaflets to develop normally, a condition that is histologically similar to myxomatous degeneration in the adult.

Index terms
  • Heart defects
  • congenital
  • Tricuspid valve insufficiency
  • Received March 1985.
  • Accepted May 1985.
  • Copyright © 1985 The Cleveland Clinic Foundation. All Rights Reserved.
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Cleveland Clinic Journal of Medicine: 52 (3)
Cleveland Clinic Journal of Medicine
Vol. 52, Issue 3
21 Sep 1985
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Severe congenital tricuspid regurgitation in the newborn
Douglas S. Moodie, Richard Sterba, Norman B. Ratliff
Cleveland Clinic Journal of Medicine Sep 1985, 52 (3) 403-408;

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Severe congenital tricuspid regurgitation in the newborn
Douglas S. Moodie, Richard Sterba, Norman B. Ratliff
Cleveland Clinic Journal of Medicine Sep 1985, 52 (3) 403-408;
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Keywords

  • Heart defects
  • congenital
  • Tricuspid valve insufficiency

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