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Session V: Insights into Neuromodulation of Cardiovascular Function

Neuromodulation of cardiac pain and cerebral vasculature: Neural mechanisms

Robert D. Foreman, PhD and Chao Qin, MD, PhD
Cleveland Clinic Journal of Medicine February 2009, 76 (4 suppl 2) S75-S79; DOI: https://doi.org/10.3949/ccjm.76.s2.15
Robert D. Foreman
Department of Physiology, College of Medicine, University of Oklahoma Health Sciences Center, Oklahoma City, OK
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  • For correspondence: robertforeman@ouhsc.edu
Chao Qin
Department of Physiology, College of Medicine, University of Oklahoma Health Sciences Center, Oklahoma City, OK
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ABSTRACT

Research using animal models has helped elucidate the neural mechanisms of angina pectoris, sensitization of cardiac nociceptive stimuli, and neuromodulation of cardiac pain and cardiovascular function. Findings over the last 2 decades include evidence of convergence of visceral-somatic input to spinothalamic cells and a major role for the vagus nerve in spinal cord processing. Stress-related glucocorticoids may manipulate amygdala function, inducing hypersensitivity to nociceptive input from the heart via central sensitization of upper thoracic spinal neuronal activity. Spinal cord stimulation may have therapeutic effects, although the underlying mechanism is unclear.

  • © 2009 The Cleveland Clinic Foundation. All Rights Reserved.
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Cleveland Clinic Journal of Medicine: 76 (4 suppl 2)
Cleveland Clinic Journal of Medicine
Vol. 76, Issue 4 suppl 2
1 Feb 2009
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Neuromodulation of cardiac pain and cerebral vasculature: Neural mechanisms
Robert D. Foreman, Chao Qin
Cleveland Clinic Journal of Medicine Feb 2009, 76 (4 suppl 2) S75-S79; DOI: 10.3949/ccjm.76.s2.15

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Neuromodulation of cardiac pain and cerebral vasculature: Neural mechanisms
Robert D. Foreman, Chao Qin
Cleveland Clinic Journal of Medicine Feb 2009, 76 (4 suppl 2) S75-S79; DOI: 10.3949/ccjm.76.s2.15
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